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Acne is a common disorder of the pilosebaceous follicles with the multifactorial pathogenesis. It typically begins in adolescence when androgen hormones stimulate the production of sebum and proliferation of follicular epidermis. The openings of hair follicules become clogged with oil secretion and corneocytes. In consequence initially invisible lesions (microcomedones) and then clinically evident comedones develop. Microcomedones and comedones are further colonized by P. acnes bacteria which promote inflamed acne lesions (papules and pustules) through the production of proinflammatory mediators, free fatty acids and porphyrins.

Acne is a common disorder of the pilosebaceous follicles with the multifactorial pathogenesis. It typically begins in adolescence when androgen hormones stimulate the production of sebum and proliferation of follicular epidermis. The openings of hair follicules become clogged with oil secretion and corneocytes. In consequence initially invisible lesions (microcomedones) and then clinically evident comedones develop. Microcomedones and comedones are further colonized by P. acnes bacteria which promote inflamed acne lesions (papules and pustules) through the production of proinflammatory mediators, free fatty acids and porphyrins.